The human brain is capable of a neural workaround that
compensates for the buildup of beta-amyloid, a destructive protein associated
with Alzheimer’s disease, according to a new study led by UC Berkeley
researchers.
The findings, published today (Sunday, Sept. 14) in the
journal Nature Neuroscience, could help explain how some older adults with
beta-amyloid deposits in their brain retain normal cognitive function while
others develop dementia.